Show simple item record

dc.creatorSánchez Castillo, Hugo
dc.creatorPaz Trejo, Diana
dc.creatorVazquéz Ramírez, Josselyn
dc.creatorZarate González, Pavel
dc.creatorMigliaro, Martin
dc.date.accessioned2015-05-19T16:21:05Z
dc.date.available2015-05-19T16:21:05Z
dc.date.issued2014-11-19 00:00:00
dc.identifier.citationhttp://revistas.ucr.ac.cr/index.php/actualidades/article/view/14131
dc.identifier.issn
dc.identifier.urihttp://hdl.handle.net/10669/12715
dc.description.abstractThroughout its evolutionary course, stress has remained as an adaptive response to stimuli that may jeopardize the integrity of an organism. Within this perspective, we can classify the stressors as psychological,physical or harmful to cardiovascular stability. However, when intense stressful events occur, there is a possibility of developing PTSD. This disorder makes use of the hypothalamic-pituitary-adrenal axis, which is commonly activated during stress and is kept activated even when the stressful stimulus has ended months ago. The consequences of this condition are observed at the neuroendocrine, neurochemical and anatomical level. This review aims to give a brief report of the neurobiology of stress, PTSD, and its implications in various structures,such as the amygdala, hippocampus and prefrontal cortex.
dc.format.extent13-20
dc.relation.ispartofActualidades en Psicología Vol. 28 Núm. 117 2014
dc.subjectStress
dc.subjectCortisol
dc.subjectTrauma
dc.subjecthippocampu
dc.subjectprefrontal cortex
dc.titleNeurobiology of Posttraumatic Stress Disorder (PTSD) and its Frontostriatal Implications: a short review
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion
dc.date.updated2015-05-19T16:21:05Z
dc.language.rfc3066en
dc.identifier.doi10.15517/ap.v28i117.14131


Files in this item

Thumbnail
Thumbnail

This item appears in the following Collection(s)

Show simple item record