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dc.creatorTruong, Hong
dc.creatorDiBello, Julia R.
dc.creatorRuiz Narváez, Edward A.
dc.creatorKraft, Peter
dc.creatorCampos Núñez, Hannia
dc.creatorBaylin, Ana
dc.date.accessioned2020-06-26T17:08:25Z
dc.date.available2020-06-26T17:08:25Z
dc.date.issued2009
dc.identifier.citationhttps://academic.oup.com/ajcn/article/89/3/920/4596766?searchresult=1
dc.identifier.issn0002-9165
dc.identifier.issn1938-3207
dc.identifier.urihttps://hdl.handle.net/10669/81221
dc.description.abstractBackground: Eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA) are associated with protection against components of the metabolic syndrome, but the role of α-linolenic acid (ALA), the metabolic precursor of EPA and DHA, has not been studied. The Δ6-desaturase enzyme converts ALA into EPA and DHA, and genetic variation in the Δ6-desaturase gene (FADS2) may affect this conversion.es_ES
dc.language.isoen_USes_ES
dc.sourceThe American Journal of Clinical Nutrition, vol.89(3), pp.920-925es_ES
dc.subjectEnfermedades cardiovasculareses_ES
dc.subjectMetabolismoes_ES
dc.titleDoes genetic variation in the Δ6-desaturase promoter modify the association between α-linolenic acid and the prevalence of metabolic syndrome?es_ES
dc.typeartículo científico
dc.identifier.doi10.3945/ajcn.2008.27107
dc.description.procedenceUCR::Vicerrectoría de Investigación::Unidades de Investigación::Ciencias Sociales::Centro Centroamericano de Población (CCP)es_ES


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