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dc.creatorCornelis, Marilyn C.
dc.creatorEl-Sohemy, Ahmed
dc.creatorCampos Núñez, Hannia
dc.date.accessioned2020-07-01T18:45:48Z
dc.date.available2020-07-01T18:45:48Z
dc.date.issued2004
dc.identifier.citationhttps://jmg.bmj.com/content/41/10/758es_ES
dc.identifier.issn1468-6244
dc.identifier.urihttps://hdl.handle.net/10669/81247
dc.description.abstractThere is growing evidence that DNA damage caused by mutagens found in tobacco smoke may contribute to the development of coronary heart disease (CHD). In order to bind to DNA many mutagens require metabolic activation by cytochrome P450 (CYP) 1A1 or CYP1A2. The objective of this study was to determine the effects of CYP1A1 and CYP1A2 genotypes on risk of myocardial infarction (MI) and whether smoking interacts with genotype to modify risk.es_ES
dc.language.isoen_USes_ES
dc.sourceJournal of Medical Genetics, Vol. 41, No. 10es_ES
dc.subjectTabacoes_ES
dc.subjectinfarto de miocardioes_ES
dc.subjectenfermedad coronariaes_ES
dc.titleGenetic polymorphism of CYP1A2 increases the risk of myocardial infarctiones_ES
dc.typeartículo científicoes_ES
dc.identifier.doi10.1136/jmg.2004.022012
dc.description.procedenceUCR::Vicerrectoría de Investigación::Unidades de Investigación::Ciencias Sociales::Centro Centroamericano de Población (CCP)es_ES


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